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Weight Loss & Metabolic

Cagrilintide

Also called cagri, AM833

Not a GLP-1 at all - an amylin analogue, which is a different hormone entirely.

Where this answer comes fromLarge human trials

Tested in large, properly controlled human trials. The ranges below are drawn from that trial data alongside what the community reports using.

The quick answer

Not a GLP-1 at all - an amylin analogue, which is a different hormone entirely. Amylin works on fullness and how quickly the stomach empties, by a separate route from GLP-1. That is why it is almost always talked about alongside semaglutide rather than instead of it: two different signals, same problem.

What it is not

Not approved anywhere on its own. The phase 3 work is on the combination.

Why people use it

Fullness and slowed gastric emptying. In the combination trials the pairing outperformed either alone, which is the whole case for it.

What people report using

These are ranges other people have reported running. They are here so you can see what has actually been done — they are not medical advice and not a recommendation for you.

Fat loss & metabolic2.4mg
How often
Once weekly
Route
subcutaneous
Evidence for this
Large human trials

Mix the 10mg vial with 2ml of bacteriostatic water. That gives 5mg/ml, so one unit on a U-100 syringe is 0.05mg and every dose on the ladder lands on a whole unit.

Work out your own units on the reconstitution calculator — the most common serious mistake in this area is arithmetic, not chemistry.

What to expect, and when

First thing people notice
Weeks 1–2 (at the table)
When it gets real
Weeks 4–6 (scale movement)
Where it peaks
Months 6–16 (still climbing)
Patience needed
High - slow and quiet by nature
The bit people get wrong

This is not a GLP-1 and it will not feel like one. There is no moment where the food noise goes silent - appetite stays, and the change shows up at the end of a meal instead of the start. Almost everyone who quits early quits because they spent two weeks waiting for a feeling that was never going to arrive. Judge it by portion size and by how long you go afterwards, not by how much you want dinner. Two other things to hold in mind: people respond to this very differently, so feeling nothing at first is common and so is being hit hard, and fatigue rather than nausea is what most often makes someone stop. And give it longer than you think - the 26-week trials showed 6-10%, the 68-week trial showed 11.8%. This one is still working long after most people have made up their mind about it.

Side effects, and what people do about them

Nausea is the main one from the trials, and it tracks the steps rather than the compound - it turns up in the days after a step up and settles. In the phase 3 trial the gut effects were mostly mild to moderate and improved over time, and only about 1% of people stopped because of nausea. If a week was rough, hold that step rather than climbing. If it is not settling, back down one.

Fatigue is the one to actually watch for, and you will not find it emphasized in the trial data. In real-world reports it is the most common reason people stop - described as being drained, flattened, no energy for a few days after the shot. If that is you, it is a known effect and not something you are imagining. Tell us rather than pushing through it.

Injection-site reactions are more common with this than with a GLP-1 - 17% against 2.6% on semaglutide in the same trial. Usually redness or a small lump. Rotate your sites and give each one a rest.

Response varies more than with most things we carry. Some people feel almost nothing for weeks; others are affected strongly and quickly. If you find yourself genuinely unable to eat rather than simply eating less, that is too much - stop and message us the same day. Not eating is not the goal and it will cost you muscle.

Go deeper

Open these only if you want them.

How it actually works

Long-acting amylin analogue.

What people report, in full

What people using cagrilintide on its own describe does not line up neatly with the trial data, and it is worth knowing before you start.

Response is split, and split widely. A fair number of people report feeling nothing at all, three weeks in. Others are hit hard and fast, describing days where a couple of bites was all they could manage. Same compound, similar amounts. There is no way to know in advance which one you will be, and that is the best argument there is for starting low and moving up slowly rather than starting where the trial did.

Fatigue is the thing the trials do not prepare you for. It comes up over and over, in separate reports from people with no connection to each other - drained, wiped out, no energy for days after the shot - and for several of them it was the reason they stopped, not nausea. It is the most consistent theme in the whole community record and it is barely visible in the published data.

The fullness-not-appetite distinction is confirmed by the people using it, and it cuts both ways. One person described being full faster while the food noise stayed exactly the same, and still being able to eat past it. If your problem is portion size, that is the compound doing its job. If your problem is thinking about food all day, this may not touch it.

The amounts people actually use run below the trial's, consistently and in the same direction. We are not publishing those numbers, because we have not counted them yet and a ladder with no number of people behind it is just a rumour in a nicer font. If you have seen them, bring them to us before you draw anything up.

It is often used as a bridge. A recurring pattern is people who have stalled on retatrutide, or who want a break from a GLP-1, running cagrilintide on its own to hold their weight in the meantime. Those reports are mostly positive. One caution from the same record: for some people the strong early effect fades and does not return at the same amount.

Sources and review

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